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Selected Highlighted News in the field of Autoimmune

Encephalitis July 2018 1st edition

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This issue includes ~ IAES has been nominated for the WEGO Health Awards 2018! Exciting Discovery of the Molecular Mechanism that causes anti-NMDAr encephalitis!  Review of Immune-mediated disorders of the CNS in children from Doctor’s on our Doctor’s list. Listings of neurology patient support groups

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IAES Nominated for WEGO Health Awards 2018


We at Int’l AE Society are proud and honored to be recognized for our advocacy and work in the Autoimmune Encephalitis Community.  The categories we have been nominated for are: 

Best in Show: Community

Best in Show: Facebook

Best Team Performance

Please share this and endorse our nomination. You can vote once in each category. 

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Exciting discovery in anti-NMDAr! A new study Directed by Dr. Dalmau, examines the molecular mechanisms that cause a type of autoimmune encephalitis: anti-NMDAr

Open Access article.
Study just released examines the mechanism that begins the disease process (parthenogenesis) of anti-NMDAr encephalitis. This will lead to Studies that will now focus on determining whether the antibodies of patients can alter other molecules that interact with NMDAR, in order to discover new potential therapeutic targets

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Neuroimmune disorders of the central nervous system in children in the molecular era

Several doctors on the IAES Doctor’s list came together in this research. Immune-mediated disorders of the CNS in children are a complex group of demyelinating, inflammatory, parainfectious and postinfectious disorders with heterogeneous pathobiological mechanisms and clinical manifestations, often associated with fundamental derangement in immune regulation. In this Review, researchers aim to provide an update on their knowledge of neuroimmune disorders and highlight areas of research that are priorities for improving clinical management.

Beyond the limbic system: disruption and functional compensation of large-scale brain networks in patients with anti-LGI1 encephalitis

Conclusions Anti-LGI1 encephalitis is associated with a characteristic pattern of widespread functional network alterations. Increased DMN connectivity seems to represent a compensatory mechanism for memory impairment induced by hippocampal damage. Network analyses may provide a key to the understanding of clinical symptoms in autoimmune encephalitis and reveal changes of brain function beyond apparent structural damage.

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International Autoimmune Encephalitis Society (IAES) is a Family/Patient centered organization that assists members from getting a diagnosis through to recovery and the many challenges experienced in their journey. 


Driven by the knowledge that “Education is Power”, Int’l AE Society manages an educational support group for patients diagnosed with Autoimmune Encephalitis and their loved ones on Face Book, empowering them to be strong self-advocates and advocates that will lead them to best outcomes and recovery. We are the premiere organization leading in these vital roles.

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Our website is not a substitute for independent professional medical advice. Nothing contained on our website is intended to be used as medical advice. No content is intended to be used to diagnose, treat, cure or prevent any disease, nor should it be used for therapeutic purposes or as a substitute for your own health professional's advice. Although THE INTERNATIONAL AUTOIMMUNE ENCEPHALITIS SOCIETY  provides a great deal of information about AUTOIMMUNE ENCEPHALITIS, all content is provided for informational purposes only. The International Autoimmune Encephalitis Society  cannot provide medical advice.

International Autoimmune Encephalitis Society is a charitable non-profit 501(c)(3) organization founded in 2016 by Tabitha Andrews Orth, Gene Desotell and Anji Hogan-Fesler. Tax ID# 81-3752344. Donations raised directly supports research, patients, families and caregivers impacted by autoimmune encephalitis and to educating healthcare communities around the world. Financial statement will be made available upon request.

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